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The Wright and Harding 2015 review in the Journal of Alzheimers Disease situated the HGF/c-Met system more broadly as a candidate cognitive-research target, summarizing the receptors roles in neurogenesis, synaptic plasticity, and neuroprotection [4]

We identified P5 in a high-throughput autocrine-based screening of large combinatorial peptide libraries, and show that P5 promotes G-protein signalling comparable to GLP-1 and Exendin-4, but exhibited a significantly reduced -arrestin response

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Research has shed light on the fact that these small molecular interactions between AR, FlnA, Rac1, and FAK trigger cell migration, driving force in PC progression and metastasis [33]