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This is due to the bacteriostatic agent, usually benzyl alcohol, that inhibits the growth of bacteria in the vial

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RSL3 stimulation elevated C3, IL-1, and TNF- levels in mixed glial cultures, while the ferroptosis inhibitor liproxstatin-1 effectively attenuated RSL3-induced neurotoxicity, supporting a role for ferroptotic stress in promoting neurotoxic glial activation

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[21] [48] [52] [53] [54] [55] [56] Deaths from chelation therapy [edit] In August 2005, a five-year-old boy with autism died while undergoing chelation therapy