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doi: 10.1371/journal.pone.0235381

The dual-incretin logic behind GLP-1 and GIP receptor synergy is also a distinctive marker

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This is only done in refractory cases that have failed all medical management (usually at least two years of treatment)

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The diverse mechanisms of action among agents such as tirzepatide, denifanstat, semaglutide, dapagliflozin, and efruxifermin highlight the complexity of MASH pathogenesis and the need for tailored treatment approaches