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The observed dynamics of these metabolites in response to supplementation and disease states offer new insights into redox biology and metabolism

However, following CaOX exposure, Fsp1 / cells exhibited extensive fragmentation, with mitochondria appearing as punctate structures (Fig

Plasma and tissue GSH levels decline with age, while markers of oxidative damage increase, creating a "redox imbalance" implicated in the pathogenesis of age-related diseases [2][3][10][11]

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